Triple-peptide protocol targeting the core hallmarks of aging
Epitalon (AEDG Peptide)
Mitochondrial Open Reading Frame
RNR2 Mitochondrial Peptide
The Longevity Research protocol addresses three core hallmarks of aging through complementary peptide interventions. Epithalon is administered in cycling protocols (typically 10-20 days on, followed by a rest period) to investigate telomerase activation without receptor desensitisation. MOTS-C is administered daily to study its effects on mitochondrial function, AMPK activation, and metabolic homeostasis. Humanin is dosed daily for cytoprotective research. All three are supplied as lyophilized vials reconstituted with bacteriostatic water. A typical 10-week protocol involves cycling Epithalon while maintaining continuous MOTS-C and Humanin administration.
PRODUCTS
3
DURATION
30 Days
PURITY
>99%
SAVINGS
15%
The Longevity Research Stack combines Epithalon, MOTS-C, and Humanin — three peptides that target distinct hallmarks of biological aging. Epithalon is investigated for telomerase activation and telomere maintenance, MOTS-C is a mitochondria-derived peptide studied for metabolic homeostasis and exercise mimetic effects, and Humanin is researched for cytoprotection and age-related cellular decline. This triple-compound protocol represents a cutting-edge approach to longevity research, addressing epigenetic, mitochondrial, and cytoprotective aging pathways simultaneously.
Each peptide targets a distinct hallmark of aging: Epithalon addresses telomere attrition (epigenetic aging), MOTS-C addresses mitochondrial dysfunction (metabolic aging), and Humanin addresses cellular stress and apoptosis (cytoprotective aging). Together, they cover more aging hallmarks than any two-compound protocol.
Published research typically administers Epithalon in cycles — commonly 10-20 consecutive days followed by a 4-6 month rest period. This cycling approach is based on the compound's mechanism of telomerase activation, where effects persist beyond the active administration period.
MOTS-C is a mitochondria-derived peptide that activates AMPK signalling, the same metabolic pathway stimulated by exercise. Research shows it improves glucose metabolism, enhances fatty acid oxidation, and may improve physical performance markers — effectively mimicking some metabolic benefits of exercise at the cellular level.
Humanin is a mitochondria-derived microprotein that declines significantly with age. It has been studied for neuroprotection (particularly in Alzheimer's models), cytoprotection against oxidative stress, and regulation of apoptosis. Its decline correlates with age-related disease progression, making its supplementation a promising area of longevity research.
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